Continuous measurement of myocardial oxygen consumption (MVO2) and hemodynamic response during transapical left ventricular bypass (TALVB)

K. Watanabe; N. Kabei; J. McRea; J. Peters

Continuous measurement of myocardial oxygen consumption (MVO₂) and hemodynamic response during transapical left ventricular bypass (TALVB)

K. Watanabe, N. Kabei, J. McRea, J. Peters

Department of Bioscience and Engineering

Research output: Contribution to journal › Article › peer-review

18 Citations (Scopus)

Abstract

Continuous measurement of hemodynamic parameters during acute TALVB and MVO₂, with the use of a fiberoptic coronary sinus catheter, has demonstrated complete removal of ventricular pulsations and reduced mean left ventricular pressure, with maintenance of the systemic circulation. Myocardial O₂ demand has been shown to significantly decrease during maximum LV decompression in the presence of a minimally changing O₂ supply.

Original language	English
Pages (from-to)	566-572
Number of pages	7
Journal	Transactions - American Society for Artificial Internal Organs
Volume	21
Issue number	1
Publication status	Published - 1975 Apr

ASJC Scopus subject areas

Medicine(all)

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title = "Continuous measurement of myocardial oxygen consumption (MVO2) and hemodynamic response during transapical left ventricular bypass (TALVB)",

abstract = "Continuous measurement of hemodynamic parameters during acute TALVB and MVO2, with the use of a fiberoptic coronary sinus catheter, has demonstrated complete removal of ventricular pulsations and reduced mean left ventricular pressure, with maintenance of the systemic circulation. Myocardial O2 demand has been shown to significantly decrease during maximum LV decompression in the presence of a minimally changing O2 supply.",

author = "K. Watanabe and N. Kabei and J. McRea and J. Peters",

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AU - Peters, J.

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AB - Continuous measurement of hemodynamic parameters during acute TALVB and MVO2, with the use of a fiberoptic coronary sinus catheter, has demonstrated complete removal of ventricular pulsations and reduced mean left ventricular pressure, with maintenance of the systemic circulation. Myocardial O2 demand has been shown to significantly decrease during maximum LV decompression in the presence of a minimally changing O2 supply.

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